Prostamax is a bioengineered active peptide designed for treating various prostate diseases.
Developed through extensive molecular-level research into the pathological mechanisms of these disorders, it incorporates partial sequences from specific growth factors, anti-inflammatione peptides, or signal-modulating peptides.
Amino acid substitutions, series truncation, and cyclization are used to enhance its stability, bioavailability, and targeting; reduction in degradation and consequently a prolonged half-life are direct outcomes of this process.
Specific action on prostate tissue is achieved.
Super, Prostamax represents a multi-mechanism, synergistic bioactive solution to improving male prostate physiological health.
Sequentia
Lys-Glu-Asp-Pro
CAS Number
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Formulae hypotheticae
C20H33N5O9
M. Pondus
487.51
Research Of Prostamax Peptide
1.Mechanisms of Action and Therapeutic Effects
Inhibiting key inflammatory signaling pathways within the prostate itself is one of its primary modes of operation – critical inflammatory factors exhibit significantly suppressed expression.
Effective alleviation of all forms of inflammatory conditions, associated swelling and subsequent pain, ensue.
Modulatio androgen receptor significat (or direct influence on 5α-reductase actio) regulates DHT (Dihydrotestosterone) productio (the more potent form of testosterone) and effectively controls epithelial cell proliferation.
Excessive proliferation caused by unregulated DHT is thus inhibited, lower urinary tract symptoms (LUTS) being a direct result of prostate enlargement.
Studies have also indicated significant potential for Prostamax in promoting apoptosis of prostate cells, angiogenesis inhibition, and comprehensive antioxidant/anti-inflammatory defense mechanisms.
2.Regulating Hormonal Pathways
Ut supra, the conversion of testosterone to DHT is pivotal to its action.
Aberrant proliferation is significantly reversed by this process.
3.Anti-inflammatio et Antioxidant Effectus
The aforementioned inflammatory and oxidative damage is mitigated.
4.Anti-Proliferative Function
Cell cycle arrest (G1 tempus) is a demonstrated effect of Prostamax treatment, and as a result of all the above, a restored physiological state of the prostate can be achieved.
Pressure on the urethra and bladder, and associated symptoms of chronic disease, are relieved.
COA
HPLC
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